Excerpt– "Crispy Ear" Syndrome...aka..Dermal Septic Necrosis/Gangrene -A.M. Henness, DVM ©1995, National Opossum Society |
General Pathogenesis:Any bacterial agent is a candidate for cause of this condition. Apparently, in dogs, it is especially prevalent with hypersensitivity reactions to certain Staphylococcus sp. Infectious sites from bites or other wounds, dental disease, or other systemic infectious processes are primary sources for bacterial spread. Serious, body-wide infection may affect the heart and valves, kidneys, liver, etc., and are all possibly well-established before one sees the peripheral effects (i.e. pinna, toes, fingers, tail).
Presentation in the Pinna:There are two forms of the condition:
Presentation in the Tail:
Presentation in the Digits:
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~UPDATE~ DERMAL SEPTIC NECROSIS-A.M. Henness, DVM©1996, National Opossum Society |
The term "crispy ear" refers to the most common form of Dermal Septic Necrosis (DSN), the proper name for this disease syndrome. The original article (please see "Possum Tales Vol 6, No.1) discusses its general pathogenesis; both describe the appearance of lesions in its various forms. This update section reinforces the necessity for early recognition and outlines effective treatment and appropriate monitoring. |
Because the opossum with DSN may appear to be "generally well", other than its lesions, caregivers, and even veterinarians (!), may be lulled into false belief: "It's OK; we can wait to deal with this," or "he looks healthy to me, these crusts are of no concern." |
This is a serious systemic infection! Only the extent and severity of infection varies from patient to patient. To avoid organ involvement/damage, or death after a short or protracted illness, one must begin appropriate antibiotic treatment ASAP! One dare not delay! And one dare not discontinue treatment prematurely! |
Diagnosis, treatment, and monitoring REQUIRES:
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Presentation: The animal with DSN may demonstrate a variety of non-specific problems/changes: appetite; urine or BM; behavior; attitude; sleep/activity cycles. The lesions typical for DSN appear at one or more cutaneous sites, in the effusive or dry form (+/- crusts), or a combination of both. In decreasing frequency, sites include: pinna, tail, nail-beds/digits, lips/nostrils, hands, feet, torso, limbs. The effusive form is always a more aggressive illness!
PROTOCOLTreatment: The "search" for the correct drug/drugs is still a potential problem where C&S is unobtainable. However, from my experience with well over 150 patients with DSN (to date), I have found the best approach is as follows, in order of "search". If one sees evidence of clinical response within 2-3 days of starting a drug/drugs, continue that antibiotic(s); if none, go to the next in order.Monitoring: physical assessment, CBC, and UA evaluated at 1-2 week intervals to verify response, 1-4 week intervals thereafter. Duration of treatment discussed at the end. |
The specific drugs listed in the protocol will not be presented on the website. Consult your veterinarian, and your membership packet. We may be available to discuss the opossum and possible treatment options with you. |
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Metabolic Bone Disease |
Please see the full text of Dr. Henness's article NUTRITIONAL METABOLIC BONE DISEASE: Its Causes . . . Its Cure, or a brief description here. |
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